Dustin (Norway)

Millions of Naira are being laundered by criminals through internet fraud, which deceives the victim with a false presentation, or hacking, which involves breaking information system security to steal financial information or data. Cybercrime, more specifically internet fraud, has become a common problem facing the world. In any case, this only occurred if the attacker was able to obtain the victim's account privileges (bank account, credit card information, or login credentials). This work presented an authentication technique that can be inexpensively implemented and used to create a secure architecture that is hard to breach. It can also reduce the amount of overhead that is placed on the authentication server during an attempt at authentication. The possibility of breaking the authentication technique was calculated using probability theory, and the mobile-based authentication algorithm was implemented using Android Studio. The result of this research shows the implementations of RatHole algorithm using android operating system which is file management android app that is being protected with RatHole algorithm and the probability of breaking down the algorithm for 1 to 10 authentication method keeps reducing by averagely 0.12. The implemented Rat-Hole on android devices also consumed 20 to 30 megabytes of data very less than memory consumption of recent research based on block chain solution which consumed 1 gigabyte to 50 gigabyte of memory.

This paper presents the findings of the first known large scale, quantitative study of the quality of IPv6 enablement of university websites. A mathematical algorithm that leverages multiple sources of data gathered from V6Sonar "agents" distributed globally across multiple locations, was used to calculate the IPv6 effectiveness of 1000 university websites.

The isolated rat heart perfused with washed platelets was used as a model to examine platelet-vessel wall interactions. Release of prostacyclin and thromboxane A2 was measured, using a cascade of smooth muscle bioassay tissues or radioimmunoassays of the stable hydration products. In hearts perfused with rabbit or human platelets, injection of sodium arachidonate caused release of both prostacyclin and thromboxane A2. In hearts perfused with aspirin-pretreated platelets, arachidonate released only prostacyclin indicating that thromboxane A2 originates largely in the platelets. Infusion of epinephrine (0.6-6 nmol/liter) through the heart potentiated arachidonate-induced release of thromboxane A2. Similar potentiation of thromboxane A2 release was observed in rat hearts perfused with either rabbit or human platelets, and in rabbit hearts perfused with rabbit platelets. In contrast, when rabbit platelets were infused through an incubation coil of tubing in place of the heart, epinephrine did not alter thromboxane A2 release. There was no significant loss of rabbit platelets on perfusion through rat hearts, and no aggregates were observed in the effluent either before or immediately after arachidonate injections, even in the presence of epinephrine. Thus, potentiation of thromboxane A2 production could not be explained by aggregation. However, it is clear from these studies that physiological concentrations of epinephrine can potentiate thromboxane A2 release from platelets when they are stimulated by arachidonic acid within the heart. This could result from a redirection of arachidonate metabolism to a local potentiating factor in the vessel wall. Potentiation of thromboxane A2 release might contribute to myocardial ischemia associated with platelet activation.