Beijing Academy of Artificial Intelligence

While spiking neural networks (SNNs) have demonstrated remarkable efficiency in neuromorphic computing by emulating biological neuronal dynamics, their learning capabilities remain constrained by predominant focus on synaptic plasticity. This limitation overlooks critical neurobiological evidence showing that intrinsic neuronal plasticity, particularly dynamic threshold adaptation, plays an essential role in balancing neural responsiveness and signal fidelity. Inspired by two neurophysiological principles governing threshold regulation: 1) the inverse correlation between spiking thresholds and preceding depolarization rates, and 2) the proportional relationship between thresholds and average membrane potentials, we propose a Membrane Potential-Driven Adaptive Threshold Plasticity (MPD-ATP) framework. This biologically grounded mechanism establishes a dual-pathway control system where instantaneous depolarization rates and sustained membrane potential states jointly modulate neuronal thresholds through an adaptive scaling factor. The instantaneous depolarization rate dynamically lowers thresholds during strong input bursts, while the sustained average membrane potential adjusts the baseline threshold to stabilize firing during sparse input. This complementary regulation improves precision and robustness. Extensive evaluations on static (CIFAR-10/100) and neuromorphic (CIFAR10-DVS, DVSGesture) benchmarks demonstrate that MPD-ATP-enhanced networks achieve superior classification accuracy with enhanced noise robustness. Systematic ablation studies reveal that the coordinated interaction between depolarization-sensitive and membrane potential-proportional threshold adjustments is critical for preventing signal saturation in high-activity networks while mitigating under-activation in sparse-input scenarios.