Comparative data on cancer in aging twins.

Description of the Genetic Analysis Workshop 10 bipolar disorder linkage data sets

Protein ScienceVolume 5, Issue 5 p. 981-981 ErrataFree Access Denaturant m values and heat capacity changes: Relation to changes in accessible surface areas of protein unfolding Jeffrey K. Myers, Jeffrey K. Myers Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this authorC. Nick Pace, C. Nick Pace Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this authorJ. Martin Scholtz, J. Martin Scholtz Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this author Jeffrey K. Myers, Jeffrey K. Myers Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this authorC. Nick Pace, C. Nick Pace Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this authorJ. Martin Scholtz, J. Martin Scholtz Department of Biochemistry and Biophysics. Department of Medical Biochemistry and Genetics, and Center for Macromolecular Design, Texas A&M University, College Station, Texas 77843Search for more papers by this author First published: May 1996 https://doi.org/10.1002/pro.5560050521Citations: 3AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article.Citing Literature Volume5, Issue5May 1996Pages 981-981 RelatedInformation

Journal of Comparative NeurologyVolume 64, Issue 2 p. 313-324 Article Lamination of the medial lemniscus in Macacus rhesus A. Ferraro, A. Ferraro Department of Neuropathology, New York State Psychiatric Institute and Hospital, New YorkSearch for more papers by this authorS. E. Barrera, S. E. Barrera Department of Neuropathology, New York State Psychiatric Institute and Hospital, New YorkSearch for more papers by this author A. Ferraro, A. Ferraro Department of Neuropathology, New York State Psychiatric Institute and Hospital, New YorkSearch for more papers by this authorS. E. Barrera, S. E. Barrera Department of Neuropathology, New York State Psychiatric Institute and Hospital, New YorkSearch for more papers by this author First published: August 1936 https://doi.org/10.1002/cne.900640208Citations: 11AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat Citing Literature Volume64, Issue2August 1936Pages 313-324 RelatedInformation

Depression and AnxietyVolume 33, Issue 5 p. 357-358 Commentary PSYCHOLOGIES OF SMALL DIFFERENCES John C. Markowitz M.D., Corresponding Author John C. Markowitz M.D. Professor of Clinical Psychiatry, Columbia University, Research Psychiatrist, New York State Psychiatric Institute, New YorkCorrespondence to: John C. Markowitz, M.D., New York State Psychiatric Institute, 1051 Riverside Drive, Unit #129, New York, NY 10032. E-mail: jcm42@cumc.columbia.eduSearch for more papers by this author John C. Markowitz M.D., Corresponding Author John C. Markowitz M.D. Professor of Clinical Psychiatry, Columbia University, Research Psychiatrist, New York State Psychiatric Institute, New YorkCorrespondence to: John C. Markowitz, M.D., New York State Psychiatric Institute, 1051 Riverside Drive, Unit #129, New York, NY 10032. E-mail: jcm42@cumc.columbia.eduSearch for more papers by this author First published: 26 April 2016 https://doi.org/10.1002/da.22488Citations: 2 The author has no formal conflicts of interest in writing this commentary—but, as he suggests in this commentary, we all have intellectual conflicts of interest. He aspires to evenhandedness. The author thanks Barbara Milrod, M.D. for her comments on an earlier draft of this commentary. Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article.Citing Literature Volume33, Issue5FOCUS ON TREATMENTMay 2016Pages 357-358 RelatedInformation

American Journal of Medical GeneticsVolume 17, Issue 4 p. 859-860 Letter to the Editor Occurrence of neural tube defects among first-degree relatives of probands in British Columbia P. A. Baird, P. A. Baird Department of Medical Genetics, University of British Columbia, Health Surveillance Registry Division of Vital Statistics, Ministry of Health Province of British ColumbiaSearch for more papers by this authorA. D. Sadovnick, A. D. Sadovnick Department of Medical Genetics, University of British Columbia, Health Surveillance Registry Division of Vital Statistics, Ministry of Health Province of British ColumbiaSearch for more papers by this author P. A. Baird, P. A. Baird Department of Medical Genetics, University of British Columbia, Health Surveillance Registry Division of Vital Statistics, Ministry of Health Province of British ColumbiaSearch for more papers by this authorA. D. Sadovnick, A. D. Sadovnick Department of Medical Genetics, University of British Columbia, Health Surveillance Registry Division of Vital Statistics, Ministry of Health Province of British ColumbiaSearch for more papers by this author First published: April 1984 https://doi.org/10.1002/ajmg.1320170419Citations: 1AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article.Citing Literature Volume17, Issue4April 1984Pages 859-860 RelatedInformation

Clinical GeneticsVolume 62, Issue 3 p. 191-195 Hotspots B Mittal, B Mittal Department of Medical Genetics, Sanjay Gandhi Post Graduate Institute of Medical Sciences, Lucrnow, Uttar Pradesh, India. Tel.: +91 522 440 700; fax: +91 522 440 017; e-mail: [email protected]Search for more papers by this authorUB Pandey, UB Pandey Department of Medical Genetics, Sanjay Gandhi Post Graduate Institute of Medical Sciences, Lucrnow, Uttar Pradesh, India. Tel.: +91 522 440 700; fax: +91 522 440 017; e-mail: [email protected]Search for more papers by this author B Mittal, B Mittal Department of Medical Genetics, Sanjay Gandhi Post Graduate Institute of Medical Sciences, Lucrnow, Uttar Pradesh, India. Tel.: +91 522 440 700; fax: +91 522 440 017; e-mail: [email protected]Search for more papers by this authorUB Pandey, UB Pandey Department of Medical Genetics, Sanjay Gandhi Post Graduate Institute of Medical Sciences, Lucrnow, Uttar Pradesh, India. Tel.: +91 522 440 700; fax: +91 522 440 017; e-mail: [email protected]Search for more papers by this author First published: 06 September 2002 https://doi.org/10.1034/j.1399-0004.2001.620301.xRead the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat Volume62, Issue3September 2002Pages 191-195 RelatedInformation

American Journal of Medical GeneticsVolume 92, Issue 4 p. 296-297 Letter to the Editor De novo partial duplication of long arm of chromosome 13: dup(13)(q12→q14) Robert Hermann, Corresponding Author Robert Hermann hermannr@apacs.pote.hu Department of Pediatrics, POTE, Pécs, HungaryUniversity Medical School of Pecs, Department of Pediatrics, Pécs, József A. u. 7. 7623 HungarySearch for more papers by this authorG. Soltész, G. Soltész Department of Pediatrics, POTE, Pécs, HungarySearch for more papers by this authorE. Morava, E. Morava Department of Medical Genetics and Child Development, POTE, Pécs, HungarySearch for more papers by this authorG. Kosztolányi, G. Kosztolányi Department of Medical Genetics and Child Development, POTE, Pécs, HungarySearch for more papers by this authorM. Czakó, M. Czakó MTA-POTE Clinical Genetics, Research Group, Pécs, HungarySearch for more papers by this author Robert Hermann, Corresponding Author Robert Hermann hermannr@apacs.pote.hu Department of Pediatrics, POTE, Pécs, HungaryUniversity Medical School of Pecs, Department of Pediatrics, Pécs, József A. u. 7. 7623 HungarySearch for more papers by this authorG. Soltész, G. Soltész Department of Pediatrics, POTE, Pécs, HungarySearch for more papers by this authorE. Morava, E. Morava Department of Medical Genetics and Child Development, POTE, Pécs, HungarySearch for more papers by this authorG. Kosztolányi, G. Kosztolányi Department of Medical Genetics and Child Development, POTE, Pécs, HungarySearch for more papers by this authorM. Czakó, M. Czakó MTA-POTE Clinical Genetics, Research Group, Pécs, HungarySearch for more papers by this author First published: 23 May 2000 https://doi.org/10.1002/(SICI)1096-8628(20000605)92:4<296::AID-AJMG16>3.0.CO;2-0Citations: 2Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article.Citing Literature Volume92, Issue45 June 2000Pages 296-297 RelatedInformation

Journal of Inherited Metabolic DiseaseVolume 14, Issue 5 p. 851-851 Short Report Recurrent abortion and amino-acid abnormalities D. Shaw, D. Shaw Department of Medical Genetics, University of British Columbia, Vancouver, B.C., CanadaSearch for more papers by this authorP. M. Macleod, P. M. Macleod Department of Medical Genetics, University of British Columbia, Vancouver, B.C., CanadaSearch for more papers by this authorD. A. Applegarth, D. A. Applegarth Biochemical Diseases Laboratory, B.C.'s Children's Hospital, 4480 Oak Street, Vancouver, B.C., V6H 3V4 CanadaSearch for more papers by this author D. Shaw, D. Shaw Department of Medical Genetics, University of British Columbia, Vancouver, B.C., CanadaSearch for more papers by this authorP. M. Macleod, P. M. Macleod Department of Medical Genetics, University of British Columbia, Vancouver, B.C., CanadaSearch for more papers by this authorD. A. Applegarth, D. A. Applegarth Biochemical Diseases Laboratory, B.C.'s Children's Hospital, 4480 Oak Street, Vancouver, B.C., V6H 3V4 CanadaSearch for more papers by this author First published: 01 September 1991 https://doi.org/10.1007/BF01799971AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. Volume14, Issue5September 1991Pages 851-851 RelatedInformation

Medical Journal of AustraliaVolume 150, Issue 1 p. 47-48 Letters to the Editor Coincidental detection of Down's syndrome during prenatal testing for Huntington's disease D.R. Turner PhD, D.R. Turner PhD Department of Haematoiogy, Flinders Medical Centre, Bedford Park, SA, 5042Search for more papers by this authorF.A. Firgaira PhD, F.A. Firgaira PhD Department of Haematoiogy, Flinders Medical Centre, Bedford Park, SA, 5042Search for more papers by this authorE.A. Haan FRACP, E.A. Haan FRACP Departments of Medical Genetics and Cytogenetics, Adelaide Children's Hospital, North Adelaide, SA, 5006Search for more papers by this authorD.F. Callen PhD, D.F. Callen PhD Departments of Medical Genetics and Cytogenetics, Adelaide Children's Hospital, North Adelaide, SA, 5006Search for more papers by this author D.R. Turner PhD, D.R. Turner PhD Department of Haematoiogy, Flinders Medical Centre, Bedford Park, SA, 5042Search for more papers by this authorF.A. Firgaira PhD, F.A. Firgaira PhD Department of Haematoiogy, Flinders Medical Centre, Bedford Park, SA, 5042Search for more papers by this authorE.A. Haan FRACP, E.A. Haan FRACP Departments of Medical Genetics and Cytogenetics, Adelaide Children's Hospital, North Adelaide, SA, 5006Search for more papers by this authorD.F. Callen PhD, D.F. Callen PhD Departments of Medical Genetics and Cytogenetics, Adelaide Children's Hospital, North Adelaide, SA, 5006Search for more papers by this author First published: 01 January 1989 https://doi.org/10.5694/j.1326-5377.1989.tb136335.xCitations: 1AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article.Citing Literature Volume150, Issue1January 1989Pages 47-48 RelatedInformation

Luft and 0livecrona.s~ 4 Pearson et aL,@ and Ray and Pearson.

Life Inside: A Memoir • Overcoming OCD and Schizophrenia With God in My Life • The Heart Too Long Suppressed: A Chronicle of Mental Illness

High levels of high sensitive C-reactive protein (hs-CRP), homocysteine (Hcy) and oxidative stress are known to be associated with premature vascular disease in type 2 diabetes mellitus (DM). The aim of this study was to estimate homocysteine levels and oxidant-antioxidant status and to determine the relationship between them in type 2 diabetic patients with and without microalbuminuria. The study population consisted of 75 subjects [age and sex-matched] divided into three groups. Fasting blood samples were obtained from 50 diabetic patients (25 with and 25 without microalbuminuria) and 25 healthy subjects. The level of blood glucose, HbA1C, urea, creatnine and serum lipids were significantly higher in the patients with microalbuminuria compared with patients without microalbuminuria and control subjects, comparatively higher level in patients with microalbuminuria. On the other hand HDL-C was found to be significantly lower in patients with microalbuminuria. The level of hs-CRP and Hcy levels were found to be significantly higher in patients with microalbuminuria compared with patients without microalbuminuria and healthy controls. The level of plasma thiobarbituric acid reactive substances (TBARS) was markedly increased and the level of enzymic and non-enzymic antioxidant was significantly decreased in the patients with microalbuminuria compared with patients without microalbuminuria and control subjects. The present study highlights the occurrence of lipid peroxidation and possible breakdown of antioxidant status in patients with microalbuminuria. Decreased antioxidant levels, increased lipid peroxidation and increased hs-CRP and Hcy levels were observed in patients with microalbuminuria.

On Sibling Designs

Journal Article Experimental Allergic Encephalomyelitis Induced by Brain Fractions Containing Mitochondria Get access L. Roizin, M.D., L. Roizin, M.D. †Dept. of Neuropathology, New York State Psychiatric Institute and Dept. of Pathology, College of Physicians and Surgeons, Columbia University, New York. Search for other works by this author on: Oxford Academic PubMed Google Scholar M. Berger Wechsler, Ph.D. M. Berger Wechsler, Ph.D. ‡Dept. of Experimental Psychiatry, New York State Psychiatric Institute and Dept. of Biochemistry, College of Physicians and Surgeons, Columbia University, New York. Search for other works by this author on: Oxford Academic PubMed Google Scholar Journal of Neuropathology & Experimental Neurology, Volume 18, Issue 4, October 1959, Pages 636–644, https://doi.org/10.1097/00005072-195910000-00016 Published: 01 October 1959

Back to table of contents Previous article Next article Clinical & Research NewsFull AccessEthnic Variations in Depression, Suicide Rates Puzzle ResearchersJoan Arehart-TreichelJoan Arehart-TreichelSearch for more papers by this authorPublished Online:5 Oct 2001https://doi.org/10.1176/pn.36.19.0025Are certain American ethnic groups more susceptible to major depression than others? The answer is yes, according to a study conducted by Maria Oquendo, M.D., a psychiatrist at the New York State Psychiatric Institute, and her colleagues and reported in the October American Journal of Psychiatry.Oquendo and her colleagues gathered data about the depression status of various American ethnic groups from two surveys that had been conducted during the 1980s, the Epidemiological Catchment Area Study, conducted by the National Institute of Mental Health, and the Hispanic Health and Nutrition Epidemiologic Survey (HHNES), conducted by the National Center for Health Statistics. The investigators used the one-year prevalence rates of major depression for African Americans, whites, and Hispanics in Los Angeles from the ECA study, which was conducted in five sites in the United States. The investigators used one-year major depression rates for Puerto Ricans on the mainland United States, Mexican Americans, and Cuban Americans gathered by the HHNES in geographic areas where these Hispanic groups were most concentrated.The one-year prevalence rates for major depression, Oquendo and her team found, were 2.5 percent for Cuban Americans, 2.8 percent for Mexican Americans, 3.5 percent for African Americans, 3.6 percent for whites, and 6.9 percent for Puerto Ricans living on the mainland United States.When Psychiatric News asked Oquendo whether these results, which are based on 1980s data, apply to the present, she replied: “That is a very difficult judgment. Obviously they are dated information. . . . So I am not sure we can extrapolate directly about what’s happening today.” However, if the findings are still relevant today, one can conclude that Cuban Americans and Mexican Americans have a modestly lower prevalence of major depression than whites and African Americans, yet the prevalence of major depression among Puerto Ricans living on the mainland United States is about double that for whites and African Americans.The reasons for these similarities and differences are not so apparent from the study. One possible explanation, the researchers suggest in their study report, is that “since the rates of major depression in Puerto Rico have been shown to be the same as in the general U.S. population, perhaps the migratory process selectively involves Puerto Ricans with higher levels of psychological distress.”Even more baffling is why the prevalence of major depression among the various ethnic groups was not always found to coincide with the prevalence of suicide among the same groups, since major depression is well known to be a significant contributor to suicide.Five ethnic groups studied by researchers at the New York State Psychiatric Institute had notable differences in their rates of suicide relative to one-year prevalence of major depression.The investigators used 1986-88 suicide data from the National Center for Health Statistics to determine the annual suicide rate relative to the one-year prevalence of major depression among the five ethnic groups and found that the rate was highest for whites, followed by Cuban Americans, African Americans, Mexican Americans, and finally Puerto Ricans on the mainland. This was the case for both men and women in each ethnic group, although suicide rates were higher among men in each group (see table).When Psychiatric News asked Oquendo why depressed Mexican Americans and depressed Puerto Ricans on the mainland appear to be especially buffered against suicide, she replied: “It may have to do with cultural issues such as relationship with the family and the extended support network, but also with the concept of fatalism. This concept is prevalent in these two cultures. The concept is that things will not necessarily go smoothly—you kind of expect that adversity will occur, and you have to tolerate it and deal with it. I think religion may be related to that as well. But these are just speculations on my part because we didn’t have data to analyze that.”However, such data may be forthcoming, she indicated, since “we are currently doing a study looking at reasons for living among the Hispanics compared with non-Hispanics to see whether we can identify the particular things, at least cognitively, that might keep Hispanic people further from suicide, or protect them, if you will.”Identifying psychological factors that protect people against suicide is a major goal of their research, Oquendo indicated.“Ethnic and Sex Differences in Suicide Rates Relative to Major Depression in the United States” is posted on the Web at ajp.psychiatryonline.org under the October issue. ▪ ISSUES NewArchived